Bio-Techne Announces Travel Grants to Attend AACR2017

Dimple Bhatia, Clinician Lab Scientist
Mumbai, INDIA

Shujie Yang, Research Assistant Professor
Iowa City IA, USA

Darina Siegmund, Graduate Student
Tübingen, GERMANY

Keith Rennier, Postdoctoral Research Scholar
St. Louis MO, USA

Marta Castiglia, Post Doctoral Fellow
Palermo, ITALY
Gagan Chhabra, Research Associate
Madison WI, USA

Tanuka Biswas, Post Doctoral Fellow
Stanford CA, USA

Sofia Karkampouna, Post Doctoral Fellow

Nathaniel Boyd, Graduate Student
Birmingham AL, USA

Donghwa Kim, Research Assistant

Terms and Conditions:

  1. Travel grants will be awarded to support attendance at AACR 2017 held in Washington DC, April 1-5, 2017 and no other event.
  2. All applications must be submitted to Bio-Techne via the online application between Dec 7, 2016 and Feb 16, 2017.
  3. Any researcher from an academic or non-profit institution is eligible to apply (technicians, PhD/graduate students, post-docs, principal investigators).
  4. Authorship on an abstract at AACR 2017 is not required.
  5. Applicants must complete the application form in full.
  6. Travel grants will not be awarded to employees of for-profit organizations (i.e. biotech or pharmacological companies).
  7. Travel grant recipients will be determined via a random drawing by Bio-Techne.
  8. The recipients will be contacted by email on or before Feb 23, 2017 and also announced online.
  9. Travel grants will be paid to the laboratory of the award recipient's principal investigator via the parent university or institution. If paid directly to the PI of the lab, taxes may apply.
  10. Proof of attendance at AACR 2017 will be required prior to travel grant payment.
  11. By submitting this application, you agree to receive information from Bio-Techne brands (R&D Systems, Tocris Bioscience and Novus Biologicals), including distributors. Your information will never be sold or used by a third party. See our complete Privacy Policy for more information.


Travel Grant  Application for  AACR 2017

My research focus on cell and molecular biology based investigations of the expression, characterization and function of nuclear receptors, hormonal regulation, adhesion molecules and signaling pathways molecules in respect to their role and participation in tumorgenesis and cancer progression particularly in case of prostate, breast and cervix cancer. In parallel I organized and have been leading the group mostly involved in cell and molecular biology based investigations of the expression, characterization and function of adhesion molecules and signaling pathways molecules in respect to their role and participation in tumorgenesis and cancer progression I have also been successfully developing the application of the novel method,  Atomic Force Microscopy, to characterization of elastic and adhesive properties of cancer cells. 

During the last 10 years, my research interest was mainly focused on the histopathology of lung tumors, molecular and cell signaling, beyond the determination of the role of specific molecules involved in the mechanisms of both intrinsic and acquired resistance to chemotherapeutic agents used in the treatment of pulmonary tumors, like anti-folate drugs and tyrosine kinase inhibitors. I employed both translational and basic research experimental approaches to improve the current therapeutic strategies and to provide the rationale for new successful drug combinations. In the last part of my Ph.D training, I developed a growing interest in fundamental aspects of molecular and cell biology also in malignant pleural mesothelioma cancer research, with promising results recently published. Preclinical encouraging data to enhance specific drug-efficacy stimulate the challenge of my future project of combining investigation in immortalized cell lines and innovative groundbreaking technologies to analyze drug-induced molecular changes in a simulated “tumor-physiological” environment: this approach aims to overcome both the lack of reliable preclinical models and poor characterization of the involved molecular mechanisms.


I’m a PhD student at University of Milan and my research activity is focused on the understanding of the role of the vacuolar proton pump V-ATPase in sustaining the stem cell niche in human glioblastoma.

My research focuses on finding novel targets in castration resistant prostate cancer (CRPC). We found the UGT2B17 enzyme expedites the CRPC progression through analysis from patient cohorts and cell/xenograft models. We propose that combination of antiandrogens with UGT2B17 inhibition could more potently suppress CRPC progression. Attending the meeting allows me to introduce the novel target to the worldwide cancer researchers and to seek collaborations to develop inhibitors. I see myself continuing to investigate the molecular mechanisms and therapeutic approaches for treating cancer patients, preparing to become an independent investigator.

I'm a young group leader in cancer research, in Peru. We conduct translational research and computational biology with a main focus on tumor biology and etiology. We are particularly interested in infection-associated cancer and the different mechanisms displayed by certain pathogens to promote carcinogenesis. For scientist in developing countries, AACR Annual Meeting is a unique opportunity to enrich our knowledge and skills as well as to speak with experts, to share ideas with them and to look for estrategic collaborations at international level. 

The concer research program in pharmacology, oncology and Molecular genetics at University Colorado Hospital   perfectly fits my academic interests with regards cancer research opportunities.  The future development of pharmacology and medicinal chemistry requires a strong collaboration between scientists and clinical applications.  I believe that pharmacology program would provide me education and resources needed to succeed in this field. With courses that span traditional chemistry and Biophysics core subjects, electives that allow for study at the pharmacology interface, and strong research programs in Molecular and Biochemistry, cell analysis, and oncology researches builds well upon my work as a student in undergraduate in Biophysics and chemistry at University of Colorado Denver. 

While many areas of pharmacology interest me. I am most impressed with pharmacology field drug composition, properties synthesis, signal transduction, molecular diagnostics and interactions toxicology.  It is made more interested when I work in the research of Molecular Genetics and Biophysics emphasizing on metabolomics by using different research techniques.  I been doing in my research principal data analysis by using MEVAN program identification and quantification on the complete set of metabolites in a biological system. Quantitative measurement of the dynamic metabolite response of living system to pathophysiological stimuli or genetic modification.  Provides important insights into physiological and diseases states and facilitate in depth of understanding of underling biochemical pathway.  I try to do with MEVAN program to analysis of metabolomics data using separation techniques. Those separation techniques make facilitate the cell to extract under Gas chromatography, capillary electrophoreses, high performance liquid chromatography and ultra-performances liquid chromatography. I work in the research in my chemistry department while I am in school. Experienced a research analysis on the biochemical effects of cyanide, where I discovered the various effects during chronic and acute period of cyanide poisoning: the metabolic effect, the effect on the endocrine system, the effect on the vascular system, the cardiac effect, the effect on the central nervous system, the effect on the pulmonary system, visual and the autonomic system, the dermatological effect. In addition to these research works, I had in my final year of studying chemistry as a major course, which I single-handedly conducted, was, analyzing the effects of Sodium artesunate in coadministration with Vitamin E on the superoxide dismutase activity in rats’ Brain and Heart. The ability of Sodium artesunate to generate reactive oxygen species known for causing oxidative damage, and the ability of Vitamin E to scavenge such reactive oxygen species were established. I did experiment by using spectrometric methods based on electromagnetic radiation, which is type energy that takes several forms as light and radiant heat. I used spectroscopic instrument based on six phenomena absorption, fluorescence, phosphorescence, scattering, emission and chemiluminescence. Optical atomic spectrometry used to identify the elements present in samples of matter and determine their concentration.

I believe my previous academic work both at the Bachelors (Biophysics and chemistry) represents my ability to thrive in challenging academic programs. As an undergraduate Majoring in Biophysics and Chemistry I spent three years’ research how ultrasounds could be used to examine arteries under pulsatile blood flow in attempt to map viscoelasticity and aid in diagnostics of vascular disease. While time constraints surrounding my graduation and difficulties in procuring approval for biological specimens in habited the advancement from theory to experimentation needed for publication the experience took me outside of the scope of traditional curricula and I gained a knowledge base of topics as varied as pulsatile flow to imagery analysis.  

My conclusion was a recommendation, to always take Vitamin E when taking Sodium artesunate, the fastest anti malaria drug, to reduce the risk of oxidative damage. I worked at Medical Oncology Neurology at school of Medicine in University of Colorado Denver Anschutz Medical campus research laboratory for three years’ career. I am working right now as internship position for pharmacology as cell analysis researcher which is more even made interested me to continue my education in pharmacology

Merkel cell carcinoma (MCC) is a rare, but aggressive form of skin cancer with rising incidence and high mortality rate. Approximately 80% of MCC tumors are positive for Merkel cell polyomavirus (MCPyV), suggesting a causative relationship between MCPyV and MCC. Exosomes are 30–150 nm vesicles that contain proteins, lipids, mRNAs, lncRNAs, and miRNAs. Exosomes from virus-infected cells comprise also viral nucleic sequences and proteins. Because tumor cell-derived exosomes may contribute to cancer, we hypothesized that MCPyV may affect the composition of exosomes and play a role in tumorigenesis. Therefore, we compared the proteins in exosomes produced by MCPyV-positive MKL1 and MKL2 and MCPyV-negative MCC13 and MCC26 MCC cell lines.  

Our proteomic analysis of exosomes originating from polyomavirus-negative and polyomavirus-positive MCC cell lines revealed the presence of the oncogenic proteins periostin and thrombospondin. Western blot analysis of exosomes and lysates of these MCC cells confirmed the presence of these proteins in exosomes from all cell lines. Thrombospondin, but not periostin was detectable in cell lysates, and an enrichment of both proteins was detected in exosomes of all cell lines. The effect of MCPyV large T-antigen (LT-ag) and small t-antigen (st-ag) on the periostin and thrombospondin promoters was examined by transient transfection studies with luciferase reporter plasmids. Transfection experiments in MCC13 revealed that LT-ag, but not st-ag significantly increased the activity of the thrombospondin and periostin promoters.  In MCC26 cells, neither LT-ag nor st-ag had a significant effect on the thrombospondin promoter activity, while both proteins alone or in combination significantly stimulated the periostin promoter strength. Our results suggest that MCPyV proteins may contribute to tumorigenesis by enhancing the expression of the oncoproteins thrombospondin and periostin and promote their secretion via exosomes.

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