Inhibitor of actin polymerization. Inhibits cell division, migration and glucose transport. Causes G2
/M cell cycle arrest and apoptosis in HCT-116 colorectal carcinoma cells. Cell permeable.
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Cytochalasin B may shorten actin filaments by a mechanism independent of barbed end capping.
Theodoropoulos et al.
Compartmentalization of transport and phosphorylation of glucose in a hepatoma cell line.
Whitesell et al.
Changes in subcellular localization of visfatin in human colorectal HCT-116 carcinoma cell line after cytochalasin B treatment.
Buldak et al.